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The pituitary gland itself remains active and engaged, potentially preventing the atrophy that occurs with exogenous hormone replacement

Within that hypothetical framework, patients with acute decompensation or an ACLF-like phenotype could be conceived of as experiencing a burst of reactive oxygen species generation that overwhelms endogenous antioxidant defences, with exogenous glutathione repletion mechanistically plausible during a transient window of vulnerability

no completed Phase 3 trials with GHK-Cu as the primary intervention

The cysteine in whey protein is largely present as glutamylcystine, which is resistant to digestive degradation and serves as an efficient substrate for glutathione synthesis [22]