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For the double and triple combinations, the abundance in phosphorylation marks suggested that mainly kinases linked to cell cycle and cell proliferation are differentially regulated (UL97, PRKD1, CDK2, CDK4) ( Figures 6C, D )

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This mechanism involves: Control of actin filament assembly and disassembly Enhanced cellular migration through cytoskeletal reorganization Improved cell morphological changes during repair processes Regulation of cellular mechanical properties The peptides actin-binding domain (LKKTET sequence) allows precise modulation of cytoskeletal dynamics, enabling cells to migrate more effectively to sites requiring repair