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Accordingly, production of the endogenous antioxidant glutathione (GSH) by glial cells within the brain [8], particularly astrocytes [8,9,10], is a critical function, as most antioxidants are unable to cross the blood brain barrier

Some of these claims have clinical grounding, while others stretch the evidence considerably

This dissociation between anti-inflammatory efficacy and bone-preserving capacity suggests that suppression of systemic inflammation may not be sufficient to reverse or prevent skeletal damage, especially in models with advanced joint destruction

This condition may arise from increased production and accumulation of reactive species or from reduced availability or function of antioxidant mechanisms