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This phenotype suggests that Cse / mice, even on standard chow, exhibit a basal ISR driven by Gcn2 , as evidenced by the mild increase in ISR genes such as Asns in the liver (Supplementary Table 2)

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ROS-mediated outer mitochondrial membrane permeabilization and cytochrome c (an apoptosis-inducing agent) translocation, to the cytosol begin the intrinsic mitochondrial apoptotic pathway, where they activate caspase-dependent cell apoptosis [42]