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Description

Glutathione reductase uses NADPH to turn GSSG back into GSH

First isolated by Croatian researcher Predrag Sikiri at the University of Zagreb in the 1990s

At the molecular level, ischemic AKI is driven by excessive oxidative stress, mitochondrial dysfunction, and activation of innate immune signaling cascades, including Toll-like receptor (TLR)/NF-B, JAK/STAT, PI3K/Akt/mTOR, and Sonic Hedgehog (Shh) pathways (Bai et al