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Overall, it has not yet been confirmed that GSH redox imbalance is a causative factor in PD or that PD pathways cause GSH imbalance in PD

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The concentration of GSH present in the mitochondria is kept constant due to the transport of GSH from the cytosol, through two GSH transportation systems, one of high-affinity, stimulated by ATP, and one of low-affinity, stimulated by ATP and ADP [36]