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Increased chondrocytic gene expression is associated with improved repair tissue quality and graft survival in patients after autologous chondrocyte implantation

It acts prominently on nitric oxide (NO) signaling , counteracting the damaging effects of NO inhibition (e.g., L-NAME) and modulating eNOS through the upregulation of antioxidant enzymes (e.g., HO-1)

In stem cell models, it has been associated with increased integrin expression and markers of proliferative potential, raising interest in its role in regenerative contexts