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Methods A comprehensive search was conducted across PubMed, Scopus, and Embase up to July 2024

Developed primarily for preclinical research targeting metabolic dysfunction, it elevates intracellular NAD+ pools via NNMT suppression to regulate lipid metabolism, insulin sensitivity and overall bodily metabolic function

Radi R (2004) Nitric oxide, oxidants, and protein tyrosine nitration

When NNMT is active, fat cells become metabolically lazy. By blocking it, this peptide reactivates fat cell metabolism, increases NAD+ availability in adipose tissue, and essentially flips a switch from fat-storing to fat-burning at the cellular level