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It neutralizes harmful reactive oxygen species, which are byproducts of metabolism, shielding cellular components like DNA, proteins, and lipids from oxidative stress (1)

Given the prominent role of anxiety in PTSD and specific requirement for NO to generalize extinction of fear learning to novel contexts (Sadeghi et al., 2022), PTSD is a particularly promising candidate for therapeutic NOS inhibition

A way to explain cell death associated with reduced levels of GSH is ferroptosis, a unique iron-dependent form of non-apoptotic cell death, characterized by lipid peroxidation with ROS accumulation due to GSH peroxidase inactivation and high levels of GSH consumption

Chronic tendinopathies like the rotator cuff, patellar, and Achilles respond to BPC-157