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Further research is needed in vitro to investigate whether aluminum oxalate is the activating material of TRPA1

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The activity of caspase 3, but not caspases 1, 8, or 9, is increased in the BSO-treated E47 cells, and damage to mitochondria appears to play a role in the CYP2E1- and BSO-dependent toxicity, because mitochondrial membrane potential is found to be decreased in the process [124]

What the broader literature describes is remodeling, not a one-way pile-up