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The observed plasma increase in adenosine could be due to a decrease in adenosine deaminase activity, inhibition of adenosine kinase, or an increase in 5-nucleotidase, all of which can occur in the presence of oxidative stress [7072]

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however, evaluation of clinically important primary outcomes has not shown improvement and should be prioritized in future trials

Down-regulation of the LXR transcriptome provides the requisite cholesterol levels to proliferating hepatocytes