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Consequently, therapeutic strategies that simultaneously enhance neurotrophic signaling and restore neurogenesis may offer superior and more durable antidepressant effects compared with monoaminergic modulation alone (44, 45)

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Moreover, the urinary excretion of isoprostane, a product of the nonenzymatic oxidation of arachidonic acid, and a marker of oxidative stress, was increased in vehicle-treated D 2 / mice and was almost completely normalized by etamicastat treatment (Fig