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Toxicology of paraquat and pharmacology of the protective effect of 5-hydroxy-1-methylhydantoin on lung injury caused by paraquat based on metabolomics

They found that loss of GSH was accompanied by a significant increase in brain nitric oxide synthase activity, by up to 55%

[2] The human ALS-deficiency picture reinforces the same point from the opposite direction: profoundly reduced circulating IGF-1 is compatible with near-normal growth when local tissue IGF-1 is intact, underscoring that the circulating pool and the local pool are not simply interchangeable

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