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Consequently, growth factors are released, and fibroblasts are stimulated into synthesizing new skin matrix such as collagen

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This mechanism involves: Sequestration of monomeric actin (G-actin) to prevent premature polymerization Promotion of actin polymerization into functional microfilaments (F-actin) when needed Enhanced cellular migration through cytoskeletal reorganization Facilitation of cell shape changes necessary for wound healing Research indicates TB-500 may work with the Arp2/3 protein complex to orchestrate actin polymerization, though this mechanism requires further clarification[4]

Hydroquinone and psoralens in the therapy of hypermelanosis and vitiligo