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The term doesnt actually tell us more than that: it doesnt indicate aetiology (cause), how the organ is affected, nor what outcome to expect

BPC-157 might be signaling the body to initiate these profound repair processes, but if the underlying cellular energy is depleted, the response could be sluggish or incomplete

Frequently Asked Questions What is the primary mechanism that makes BPC 157 a candidate for herniated disc research

It is caused by repeated amplification of the CAG trinucleotide in the huntingtin (Htt) gene on the short arm of chromosome 4, which leads to a prolonged polyglutamine stretch at the N-terminus of the protein