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Compared with the (partial) remission phase, the acute phase of MDD may exhibit a markedly distinct NIMETOX profile, with the former characterized by elevated immune-linked neurotoxicity levels and the latter characterized by CIRS activities focused on healing and restoring immune equilibrium

AChR antibodies block the acetylcholine receptors from being able to receive signals from the nerve to stimulate a muscular response

Either genetic or pharmacologic inhibition of TrxR1 activity induces Nrf2 without inducing oxidative stress, indicating that a part of the stress-sensing regulatory mechanism of Nrf2 is controlled by TrxR1 [49, 173176]

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