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PARP cleavage inhibition: PARP, a downstream target of caspase-3, saw its cleavage fragments reduced by 77.5%

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In vivo , experiments in xenograft mice bearing human lung adenocarcinoma cells indicated that tumor growth was restrained by 27.8% in mice treated with free DOX, 55.0% in the DOX@BRNPs group, and even as high as 71.9% in the laser-irradiated DOX@BRNPs group

We have gotten a better understanding of the pathophysiological mechanisms responsible for these disorders as a result of these cases [59, 60]