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Clinical studies have confirmed that hyperhomocysteinemia is not only an independent risk factor for the development of cardiovascular and cerebrovascular diseases, such as atherosclerosis, acute myocardial infarction, stroke, coronary artery lesions, and peripheral vascular disease, but also increases the risk of neurodegenerative disorders and tumors, and impairs the efficacy of antifolate drugs

Since GSH patrols have the cellular environment and put out oxidative free radical flames, it becomes oxidized itself and inactive, therefore transforming into GSSG

They can also accumulate the drug at site of action, imparts reduced side effects and trigger drug delivery and penetration due to very small droplet size 5 to 200 nm

In conclusion, although many reports previously evidenced the regulation of signaling pathways by GSTs in various in vitro cell systems, we demonstrate for the first time that the invalidation of Gstp1/p2 affects multiple key events of the hepatocyte cell cycle in vivo after PH