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[6] 1 in 5,000 have some form of EDS worldwide

Such mechanisms could detrimentally impair astrocyte functions either through the loss or down-regulation of essential homoeostatic functions or through the gain of detrimental effects, or combinations of both

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Cortistatin promotes this synchronization directly Homeostatic regulation -- preprocortistatin mRNA is upregulated after sleep deprivation and follows a circadian rhythm, indicating that cortistatin functions as an endogenous homeostatic sleep factor BDNF-dependent expression -- brain-derived neurotrophic factor regulates cortistatin expression in an activity-dependent manner, linking neuronal activity during wakefulness to subsequent sleep drive Cortistatin's receptor pharmacology is well-defined