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The expression profile of growth factors involved in OPC recruitment (PDGFRa, FGF2) and in OPC differentiation (IGF1, TGF1) following demyelination is altered in old mice, in which the upregulation of these factors is both weaker and delayed (Hinks and Franklin, Consistent with cell-autonomous effects, the epigenetic control of OPC differentiation into myelinating OLGs is disrupted with aging (Shen et al., 2008), and the OPCs of aged mice fail to respond to growth factors and differentiation signals (Neumann et al., 2016)