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Immune checkpoint molecules like PD-1 (programmed death-1) and CTLA-4 (Cytotoxic T lymphocyte-associated antigen-4) function as negative regulators of immune activation, and when triggered by tumor or tumor-associated cells, they suppress immune responses (105, 125128)

This includes the counteraction presented in the short term for all succinylcholine disturbances (i.e., agitation, muscle fasciculation)

Most newly developed pharmacon candidates to treat neurological diseases are not able to cross the BBB and reach their target(s) within the brain [3]

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