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Further studies are needed to confirm the actual benefit of this molecule on metabolic liver diseases and define the best route of administration and the most appropriate dosage, allowing its use in clinical practice

Specifically, RhoA disrupted mitochondrial function in ECs, leading to oxidative stress

The cells were lysed with methanol containing internal standard compounds, and then water-soluble fraction was separated by liquid-liquid extraction (chloroform: methanol: water=1: 2: 1)

TLR4 signalling in ischemia/reperfusion injury: a prospective target for connecting inflammation, oxidative stress and programmed cell death to improve organ transplantation outcomes