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Specifically, we identified the GLUT1TXNIP axis as a key molecular pathway mediating metformin-induced alterations in intestinal glucose metabolism and propose that this mechanism plays a critical role in its glucose-lowering effect

4 shows that an increase in the concentration of GSH results in a 14% increase in the release of ENZ from the nanocarrier within a 48-h period, which is 4.2-fold higher than the release in the absence of GSH

This updated philosophy is crucial for the management of T2D, emphasizing that TOD is not only caused by hyperglycemia