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But a 2025 systematic review confirmed there is still "little clinical evidence" in humans [6]

[DOI] [PubMed] [Google Scholar] 90.Swank Z., Borberg E., Chen Y., Senussi Y., Chalise S., Manickas-Hill Z., Yu X.G., Li J.Z., Alter G., Henrich T.J., et al

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Unlike native IGF-1, which is rapidly bound and regulated by IGFBPs in circulation, IGF-1 LR3 was engineered to remain active longer, making it a useful tool for researchers studying sustained IGF-1 receptor signaling rather than the brief signaling windows native IGF-1 typically produces