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Through PLGA degradation, lactate is released that can potentially provoke the proliferation of endothelial and fibroblast cells (Chereddy et al., 2013

However, unlike branded medicines, these compounds are not FDA-approved because no clinical trials have tested their safety and effectiveness

Glabridin exhibited therapeutic potential in the treatment of DN through improving renal function by repressing not only ferroptosis but also VEGF/p-Akt/p-ERK1/2 axis activation [247]