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We speculate that combining GSH precursors with HBAT caused a synergistic elevation of GSH, improvement of mitochondrial function through a mitohormesis effect, and clinical improvement in our patient

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From rat models of liver injury induced by intestinal ischemia/reperfusion, we have seen that the administration of SF markedly reduced MPO activity with a concomitant elevation of GSH levels, liver Nrf2 and HO-1 expression, and increased GPX activity [143]

While GHK-Cu is a naturally occurring peptide in human plasma, AHK-Cu is entirely lab-synthesized with a focus on hair follicles and general scalp health