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As a result, glutathione is at the forefront of mitigating quinone-derived toxicity, by enzymatically reducing redox-generated H 2 O 2 to harmless H 2 O via glutathione peroxidase, or by direct reaction and detoxification of the quinone electrophile (Fig

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[Molecular Pharmacology, 2015, vol

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Funding This study was supported by the National Natural Science Foundation of China (Nos

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