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A pooled analysis of these two randomised-controlled trials demonstrated a placebo-adjusted reduction in OFF-time of 1 h from baseline to week 12 [143], and an open-label extension showed persistent improvement of MDS-UPDR part IV motor fluctuation subscore after 100 weeks of treatment [144, 145]

demonstrated that senescent pancreatic ductal adenocarcinoma (PDAC) cells in mice activated endothelial cells through the release of SASP factors into the tumor microenvironment [39]