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In addition, as demonstrated in a study of BPC 157/tendon-healing relations [117,118], in tendon fibroblasts, BPC 157 increased the expressions of growth hormone receptors at both the mRNA and protein levels given the persistent promoting effect on the expression of growth hormone receptor as more significant up to three days after the treatment

It suppresses ECM degradation, counteracts RSL3 mediated inhibition of GPX4, increases intracellular GSH levels and the GSH/GSSG ratio, reduces MDA content, and inhibits activation of the MAPK/NF-KB pathways, thereby protecting chondrocytes from ferroptosis (156)