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These findings further supported that DET1 is essential as a bridge and that COP1 does not engage in a canonical RING-E2 interaction with Ube2e2 (Supplementary Fig

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While overexpression of BMP6 inhibited the expression of Wnt10b in HFSC, adenoviral Wnt10b treatment in telogenic HF reduced the number of BMP6-positive cells as compared to control