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Keeping the dynamic balance of cardiomyocytes plays a key role in maintaining the normal physiological function of heart (Ma et al., 2020), and the loss of cardiomyocytes caused by CD would induce and aggravate the development of HF

Catalpol promotes angiogenesis in human umbilical vein endothelial cells (HUVECs) by upregulating angiogenesis-related proteins, suggesting its potential therapeutic application in vascular diseases (Ni J

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