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The pathological features of non-neurological autoimmune neuropathies primarily involve immunopathological mechanisms associated with adaptive immune responses against antigens expressed in the nervous system, resulting in neuroinflammation

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However, CoA does not fit well within the modeled catalytic pocket, consistent with our experimental findings that ABHD11 does not hydrolyze the glutaryl-CoA thioester

Treatment of myalgic encephalomyelitis/chronic fatigue syndrome: a systematic review for a National Institutes of Health Pathways to Prevention Workshop