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Results PRL deficiency exacerbated HFD-induced hepatic lipid deposition, and central intracerebroventricular PRL alleviated hepatic steatosis in HFD-fed PRL/ and wild-type mice more rapidly than peripheral intraperitoneal PRL

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[13] Subsequent work on primary human muscle cell cultures found that the MGF-24aa-E peptide increased the proliferative lifespan and delayed senescence of satellite cells isolated from neonatal and young-adult muscle, though notably not from old-adult muscle