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Persistent bacterial infections: the interface of the pathogen and the host immune system
In addition to dampening NF-B activity, KPV has been shown to downregulate the expression of adhesion molecules on endothelial and immune cells, limiting the recruitment and infiltration of leukocytes into inflamed tissues.(7) This targeted modulation extends to the suppression of pro-inflammatory cytokines such as interleukin-8 (IL-8) and tumor necrosis factor-alpha (TNF-), thereby mitigating excessive inflammatory cascades without broadly compromising host immune competence (8)
HUBERMAN: So given, you know, at the level of the gut, somehow it's able to travel to the site of injury, recognize that something needs to be done there, in particular angiogenesis and fibroblast infiltration
Curr Probl Cardiol (2022) 47(12):101361
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