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Upon forming the CRL5 complex, it is hypothesized to ubiquitinate and degrade negative regulators of matrix metalloproteinases (MMP2/9), thereby enhancing their secretion and subsequently promoting cell migration and invasion

J Cardiovasc Pharmacol 41:880889 Kobayashi A, Kang M-I, Okawa H, Ohtsuji M, Zenke Y, Chiba T, Igarashi K, Yamamoto M (2004) Oxidative stress sensor Keap1 functions as an adaptor for Cul3-based E3 ligase to regulate proteasomal degradation of Nrf2

PhoA oxidative folding could be enhanced by the addition of exogenous GSSG in the WT and fully restored in a dsbA mutant

However, excessive inflammatory signaling leads to sustained TGF- expression, which prolongs myofibroblast survival and promotes pathological collagen accumulation [47]