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Selenium supplementation, known to enhance GPX4 activity, has demonstrated the ability to reduce oxidative stress and protect thyroid cells from ferroptotic injury (60, 185)

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Vourloumis group explored a large series of weaker zinc binding groups as potential alternatives to, e.g., the zinc binding amide function found in the 3,4-diaminobenzoic acid derivatives 47 and 48 (Tsoukalidou et al., 2019)