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He went on to conclude that even though the presence of senile plaques and neurofibrillary tangles were observed, the plaques are not the cause of senile dementia, but only an accompanying feature of senile involution of the central nervous system [7]

Upon sensing environmental stress or cellular damage, the absent in melanoma 2 inflammasome (AIM2) and NLR family pyrin domain-containing 3 (NLRP3) inflammasome assemble and recruit the inflammasome adapter protein apoptosis-associated speck-like protein containing CARD, leading to the activation of pro-inflammatory caspases [137]

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Semin Nephrol