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Increased production of reactive oxygen species (ROS) is a unifying mechanism for several risk factors that induce arteriosclerosis, endothelial cell dysfunction, and cardiac dysfunction ( E-cigarettes induce increased ROS in vitro and in vivo in endothelial cells ( The role of inflammation in the development of atherosclerosis ( Platelets from healthy volunteers exposed to e-cigarettes aerosol show an increase in the expression of globular complement protein C1q receptor (gC1qR) and calreticulin cC1q receptor (cC1qR), two proteins that are associated with the atherosclerotic events, platelet activation and aggregation

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