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Based on the current evidence, we propose that the iron-overloaded, ROS-prone milieu of EMS renders EESCs susceptible to Erastin-triggered, multi-pathway regulated cell death that integrates ferroptosis with necroptotic and pyroptotic signaling, while ferritin handling and mitophagy act as key modulators of susceptibility

However, diverse modes of cell death often have similar signaling pathways and interact with each other, such as necroptosis, apoptosis, autophagy, pyroptosis and ferroptosis

10.1017/s0033291721003792 103 van der HeeB.WellsJ

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