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A previous study has recently confirmed another mechanism by which MA reduces neuroinflammation and facilitates neural repair in rats with TBI: inhibition of the TLR4/TRIF/MyD88 signaling pathway (Cao et al., 2023)

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Clinically, this more advanced arterial phenotype may be associated with: Blood tests do not directly measure arterial stiffness or calcified plaque, but they can identify several contributors to these processes, including abnormal glucose regulation, kidney dysfunction, albuminuria, inflammation, and atherogenic lipid burden

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