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The mechanism, when it was finally established, was elegant: selenocysteine is encoded by the genetic code's stop codon UGA, but only in specific mRNA contexts where a particular structural feature (called the SECIS element) signals the ribosome to insert selenocysteine instead of stopping translation

Future trends and controversies in metabolic targeting strategies Despite the unprecedented promise of mitochondrial targeting and metabolic reprogramming therapies in CRC, several challenges remain

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