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Cell growth, controlled by sodium-hydrogen ion exchangers (NHEs), induces MIC-dependent activation of the PI3K/Akt pathway, which in turn supports the cytoplasmic localization of the cell cycle inhibitor p27 Kip1 , resulting in tumor cell proliferation [142]

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Exploiting the differential sensitivity of microglial subsets to ferroptosis may offer novel strategies for modulating neuroinflammation and slowing motor neuron loss