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118 Redox reactions at key cysteine residuesnamely, s -glutathionylation at Cys346 119 and reduction of the Cys272-X-X-Cys275 disulphidealso enhance CBS enzymatic activity

Differential guide scores in the indicated cell lines upon treatment with 20 M BSO (right)

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In addition, dimethylchrysin could mediate mitochondrial and endoplasmic reticulum apoptotic pathways by inducing ROS production, intracellular calcium ion dysregulation, and mitochondrial membrane potential loss in human endometriosis-derived cell lines